Cellular parasites

Cellular Parasite Detox

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Cellular Parasites

Cellular parasites are entities which inhabit cells. They are motile, and observably enter, exit and change host cells. They are large enough to be seen in darkfield and phase contrast microscopes.

This video footage illustrates four apparent species, which present as unique artifacts. These artifacts are much larger than viruses which would appear as a barely visible dot at these resolutions.

  • It is often possible to observe multiple species in the same individual;
  • It is also common to observe multiple parasites inhabiting the same cell;
  • Progressive pathology generally relates to the parasite conentration;
  • Symtpom variability corresponds to active balance in species population;
  • Artifact motion, and migration suggest they are pleomorphic life forms;
  • Artifact observation requires erythrocytic trauma to initiate.

Traditonally, virusues are the sole category of entity thought to generally unrecognized life forms frequently visable in blood and tissue samples.

Hemoglogin encapsulated. Moves between erythrocytes without entering serum.   Frequently identified as Babesia.
Phase Enhancement Artifact. White dot indicates presence of phase enhancing interior artifact.   Often observed in cancers and precancerous individuals.
Exiting Artifact. Shows a multi-node entity exiting an erythrocyte into serum.   Likely occurs when the present host cell becomes unsuitable as a host due to toxic, nutrient depletion, or energetic stress.
Entry Attemtping Artifact shows an apparent parasite attempting to enter erythrocytes.    
Phase Absorption Artifact. Black dot creates a field depression which appears as a Black dot indicating an electropolar interior artifact    

The Marshall Protocol

Trevor Marshall documened a second vulnerability fo these organisms. He asserts that these organisms plug the vitamin D receptor in host cells to prevent immunological recognition of the parasitized status. See the Marshall Protocol for more information.

The Marshall Protocol uses very low doses of hypertension medications, and tiny doses of antibiotics to enable immunological elimination of parasitized cells.

The Marshall protocl produces long term detoxificaiton and frequently involves a year, or more of acute recovery symptoms. The likely challenge of the protocol is the mobility of pathogens. When the immune system attacks the infested cells, motile pathogens move out and run away.

The likely response is that the elimination of "parasites" is likely limited to particular conditions which prevent the parasite from escaping from the immune system. It is likely the Marshall Protocol asserts highest efficacy with parsites having the lowest mobility, and which are more easily trapped, then eliminated, host cells.

The prolonged response likely reflects a low kill rate by the immune system..

Protocol Model

The protocol model selectively targets mobile entities cloaked inside cells and in serum.

Revici documented that cellular encapsulated entites, particularly viruses, were susceptible to certain structures of fatty acids containing odd-numbers of carbon atoms. He documented a particular ability to destroy viruses, and the cells they inhabit, using nontoxic fatty acids.

Revici implied that both the serum and the intracellular spaces hosted pathogens. His approach produces results with both encapsulated and free viral entities. It is probable that his approach may a produce an adverse effect on non-viral larger entities. This is supported by his high success rates, particularly when he combined mineralized lipids, with the anti-viral compounds.

Protocol Components

This protocol addresses the several vulnerabilites to fibroid growths:

  1. Therapeutically elevated enzyme levels;
  2. Cellular targeted nutrients which trigger transition to aerobic metabolism;
  3. Locally targeted supplements which disable glucose uptake;
  4. When possible triggering of cellular electroporation to accelerate uptake of therapeutic agents..

The recommended protocol uses supplements and suppositories, preferably intra-uterine, which create a therapeutic concentration of the elements which naturally limit fibroid development.

Heptanoic Acid in Sesame Oil 1 ml/day Titrate Asserts anti-viral and likely anti-parasitic effects on cells
Nonanoic Acid in Sesame Oil

1/2 ml/day Titrate

Blocks glucose receptors in anaerobic cell lines inhibiting absorption of preferred glucose nutrients.
Sulphur 150 3 ml/day Delivers Sulfur nutrient to anaerobic cells
Selenium 245 3 ml/day Delivers Selenium nutrient to pathological cells.
PEMF 30 min if possible Interferes with pathogen energetics. Supplies usable energy to immune system and body.

Expected Response

The protocol is a titration. Lipidic compounts tend to accumulate in pathogenic tissues. In normal tissues, these substances have no apparent effects.

The method uses small doses to gradually increase titers of anti-pathogenic compounds. The gradual increase in stress targets a managed decrease in pathogenic population.

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