This protocol addresses the treatment aspects of treatment resistant Small Cell Lung Cancer, or SCLC.

• A Case History

SCLC is an agressive tumor category which frequently metastasizes widely. Cells from primary tumors in the are carried from the lungs in blood stream and often spread to peripheral organs, frequently the brain.

Conventional treatment of SCLC typically results in diminishing response to chemotherapy. In most cases, successive rounds increasing intensity of chemo therapy are required to suppress tumor progression.

Eventually tumor resilience surpasses patient therapeutic tolerance, and tumors overwhelms the patient. Therapeutic tolerance forces discontinuance of chemotherapy, and tumors progress unhindered either by patient autoimmunity or by therapeutic agents.

Adaptive resistance to conventional treatment is the primary basis for poor prognosis in SCLC cases.

There are several primary challenges with conventionally treated SCLC:

• Persistence of highly resilient and adaptable tumor cells;
  • Vulnerable cells were eliminated by conventional therapy;
• Collateral Damage from toxic therapy:
  • Autoimmune deficiency;
  • Progressive liver and digestive system dysfunction;
  • Systemic suppression of all adaptive cell lines
  • Resulting from Accumulated systemic toxicity
  • From both cancer toxins and and toxic therapy agents.
• Initial vulnerability to cancer is unresolved
  • Toxicity
  • Immune Underperformance
  • Pathogenic involvement

Click here for a complete discussion of Malignancy Cofactors. This protocol is a an adapted version of the Malignancy Cofactors Protocol.

Cellular Detoxification

This detox protocol supports restoration of cellular functions inhibited by use of toxic therapeutic substances and energetics. Preferential use of cellular oxidative agents attached to glucose molecules installs plentiful toxins deeply into many if not most categories of cells which utilize glucose as an energy source.

This protocol segment utilizes a series of nutrients to mobilize and eliminate deeply embeded cellular toxins. The duration of this segment of the protocol is 8 weeks.

Energetic Support

Energetic support is a key factor in limiting propogation of tumor cells:

• Restoring Immunological Energetics enables immune cells to perform better;
• Metabolic, particularly liver energetic support helps the liver eliminate accumulated agents and those released by breakdown;
• Energetics to tissues adjacent to tumor cells helps these cells resist caustic chemical attack. This resistance helps to nutrients available for tumor cell propagation;
• Energetics applied to tumor cells reduce the rate of propagation by increasing the transmembrane potential which when low, drives cellular reproduction;
• In some cases pulsed fields have been documented to trigger apoptosis in tumor cells - this is not a consistent response, and depends on the method of application, the tumor cell line, and other other unknown variables;;
• Enhances celluar absorption of therapeutic agents. PEMF at high levels triggers electroporation which causes cells to enter enhanced respiration for about 3 hours.

Tumor Management Plan-A

These tumor management plans are organized by three criteria:

• Degree and quality of supporting published and experience data;
• Cost and convenience;

This is a tumor suppression protocol using complementary approach. It is a combination of protocols which are individually highly effective with most tumor lines.

This is an at-home protocol which uses supplements and strategies which are available in orally.

Tumor Management Plan-B

These protocols utilize large quantities of substances which are limited to IV administration.

Tumor Management Plan-C

These protocols utilizes adminstration of high amounts of selenium which selectively accumulate in tumor cells..